Postdoctoral Fellow, Antifungal Immunity

Job Title: Postdoctoral Fellow, Antifungal Immunity

Job Number: 69217

Location: Torrance, US

Job Description

A postdoctoral position is available in the laboratory of Dr. Marc Swidergall to investigate mucosal antifungal immunity.  The lab focuses on cellular and molecular factors that regulate the innate and adaptive immune responses against mucosal and invasive fungal infections.  The fellow will have the opportunity to lead their own projects and will join a dynamic and collaborative biomedical research environment that prioritizes mentoring and training. 

Dr. Swidergall is Assistant Professor at the David Geffen School of Medicine at UCLA, Division of Infectious Diseases, Harbor-UCLA Medical Center, and Investigator at The Lundquist Institute at Harbor-UCLA Medical Center, Institute for Infection and Immunity.  The dual affiliation will provide the fellow with access to numerous research and training resources from both institutes including state-of-the-art core facilities. The Swidergall lab is located on the Harbor-UCLA campus.

Qualifications

Ph.D. or equivalent in immunology, microbiology, or related fields received within the last 3 years.  Candidates should be motivated and creative, have a strong background in cellular immunology. Proficiency in cell culture techniques, standard microbiology and molecular techniques (e.g., ELISA, Western blot, PCR, flow cytometry), and animal models is strongly encouraged.  Candidates must also possess strong oral and written communication skills and have a record of publishing their research in peer-reviewed journals.  The fellow will be expected to work well both independently and as part of a diverse team in a creative and collaborative research environment.

How to apply

Interested applicants should email a cover letter describing your research interests and goals (~1 page), a curriculum vitae, and two references to Marc Swidergall ([Please click the Apply button for the link or email]).

References

Millet, N. et al. IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis. Nat Commun 13, 5545 (2022).

Swidergall, M. & LeibundGut-Landmann, S. Immunosurveillance of Candida albicans commensalism by the adaptive immune system. Mucosal Immunol 15, 829-836 (2022).

Millet, N., Solis, N. V. & Swidergall, M. Mucosal IgA Prevents Commensal Candida albicans Dysbiosis in the Oral Cavity. Front Immunol 11, 555363 (2020).

Swidergall, M. et al. EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection. Cell Rep 28, 423-433 e5 (2019)

Swidergall, M., Solis, N. V., Lionakis, M. S. & Filler, S. G. EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans. Nat Microbiol 3, 53-61 (2018).

Application Deadline: 2023-12-31